Gutersohn A, Caspari G, and Erbel R. Upregulation of VEGF-mRNA in human umbilical vascular endothelial cells via shock waves. Presentation; Eur J Heart Failure 2000;2(Suppl1):42. 

• Human umbilical vascular endothelial cells were placed into the focus of shock waves at energy flux densities of 0.05, 0.1 and 0.3 mJ/mm2 (“modified lithotripter”). After shock wave treatment cells were grown for 24-36h, and tested for VEGF-mRNA. Esw treated cells revealed a significant increase of VEGF-mRNA versus controls. Cell death increased with increasing esw energy levels.

Haupt G, and Chvapil M. Effect of shock waves on the healing of partial-thickness wounds in piglets. J Surg Res 1990;49:45-48. 

• Re-epithelialization of wounds inflicted in normal or previously irradiated skin of piglets was significantly enhanced by low energy shock-wave treatment (Experimental Lithotripter XL1, Dornier, 1000 pulses at 14 or 18 kV energy level). Shock waves of intermediate (or high) energy were without effect. 
  

Kamelger FS, Djedovic G, Meirer R, and Piza-Katzer H. Deep partial thickness burn injury and the effect of eswt. An experimental investigation in rats. Presentation no. 65; 8th Int Congress of the ISMST, May 29th to June 1st 2005, Vienna, Austria. 

• Standardized deep partial thickness burns were applied to the backs of 30 rats. Immediately after burns the esw treatment (0.11 mJ/mm2, 500 pulses) was applied on selected areas, whereas the remainder of the burns served as untreated controls. Re-epithelization was improved significantly at days 5 and 15, wound closure over time was enhanced as well (both, p < 0.05). 
  

Meirer R, Kamelger FS, Huemer GM, Wanner S, and Piza-Katzer H. Extracorporeal shock wave may enhance skin flap survival in an animal model. Presentation no. 59; 8th Int Congress of the ISMST, May 29th to June 1st 2005, Vienna, Austria. 

Meirer R, Kamelger FS, Huemer GM, Wanner S, and Piza-Katzer H. Extracorporeal shock wave may enhance skin flap survival in an animal model. Br J Plast Surg. 2005;58(1):53-57.